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Tachyarrhythmias

Torsades de pointes

Polymorphic VT twisting around the baseline, on a background of a long QT.

~250 bpmPolymorphic, self-terminating
II
II. The axis rotates so the complexes wax and wane about the baseline — the 'twisting of the points'.

What you see

  • Rapid polymorphic ventricular tachycardia whose amplitude waxes and wanes in spindles.
  • The QRS axis twists around the isoelectric line every 5–20 beats.
  • The preceding sinus beats show a prolonged QTc, often with prominent U waves.
  • Frequently initiated by a short–long–short RR sequence.

Why it looks like that

Delayed repolarisation lengthens the QT and allows early afterdepolarisations to reach threshold. These trigger re-entry in tissue whose refractory periods now vary widely across the ventricle, so the activation wavefront meanders and the axis rotates.

What to do

  • Magnesium sulphate 2 g IV, regardless of the serum magnesium.
  • Stop every QT-prolonging drug and correct potassium and calcium.
  • Increase the heart rate to shorten the QT — isoprenaline or overdrive pacing at 90–110.
  • Defibrillate if it degenerates into ventricular fibrillation.

The trap

Amiodarone is the wrong reflex here: it prolongs the QT further. And in congenital long QT, isoprenaline is contraindicated — beta blockade is the treatment. Establish whether the long QT is acquired or congenital before choosing.

magnesiumlong QTearly afterdepolarisationoverdrive pacing

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